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Statistical power is the probability that a statistical test will correctly reject a false null hypothesis (H0​) when a specific alternative hypothesis (H1​) is true. H0​ is the null hypothesis, which states there is no effect or no difference. H1​ is the alternative hypothesis, which states there is a real effect or difference. Alpha (α) is the probability of a Type I error (a false positive), which is the risk of incorrectly rejecting the H0​ when it is actually true. You set this value before the experiment, commonly at 0.05. Beta (β) is the probability of a Type II error (a false negative), which is the risk of failing to reject the H0​ when it is actually false.
Power is calculated as 1−β. Increasing power means you are decreasing the probability of making a Type II error.
Several factors can be adjusted to increase the power of a statistical test:
Effect Size: This is the magnitude of the difference you are trying to detect. A larger effect size is easier to detect, thus increasing power.Â
Sample Size: The number of observations in a study. A larger sample size provides more information about the population, reducing the margin of error and increasing the power to detect a true effect.
Variation: Refers to the spread or standard deviation of the data within the population. Less variation makes it easier to distinguish a real effect from random noise, thereby increasing power.
Alpha (α): Increasing the alpha level (e.g., from 0.05 to 0.10) also increases power, but at the cost of a higher risk of a Type I error. This trade-off is often undesirable.

Vollset, S E; Nygârd, O; Refsum, H; Ueland, P M
Coffee and homocysteine Miscellaneous
2000, ISSN: 0002-9165.
@misc{pmid10648251,
title = {Coffee and homocysteine},
author = {S E Vollset and O Nygârd and H Refsum and P M Ueland},
doi = {10.1093/ajcn/71.2.403},
issn = {0002-9165},
year = {2000},
date = {2000-02-01},
journal = {Am J Clin Nutr},
volume = {71},
number = {2},
pages = {403--404},
keywords = {},
pubstate = {published},
tppubtype = {misc}
}
Schneede, J; Refsum, H; Ueland, P M
Biological and environmental determinants of plasma homocysteine Journal Article
In: Semin Thromb Hemost, vol. 26, no. 3, pp. 263–279, 2000, ISSN: 0094-6176.
@article{pmid11011844,
title = {Biological and environmental determinants of plasma homocysteine},
author = {J Schneede and H Refsum and P M Ueland},
doi = {10.1055/s-2000-8471},
issn = {0094-6176},
year = {2000},
date = {2000-01-01},
journal = {Semin Thromb Hemost},
volume = {26},
number = {3},
pages = {263--279},
abstract = {This article gives an overview over common physiological, lifestyle, and pathological conditions that may modulate the homocysteine status. The interplay of several environmental factors, including age, gender, nutrition, smoking, and coffee consumption and physical activity with commonly used drugs and prevalent diseases are described. In most cases, an abnormal homocysteine status is not caused by a single factor alone but often is the result of combined effects. We address these frequently found "clusters" of homocysteine-modulating factors. Finally, we give an overview of likely causes of hyperhomocysteinemia found in an authentic material. This material is based on 2462 routine measurements of plasma total homocysteine carried out at the Haukeland University Hospital. The data represent the total number of combined homocysteine and methylmalonic acid determinations, requested by general practitioners in Norway during February 1998.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
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